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New Harvard Study Discovers Concerning Information About Antibiotics
infowars.com Alexander Fleming discovered penicillin in the 1940s, and it has been used as a poster child for ‘safe’ antibiotics ever since. Fleming’s discovery heralded the ‘age of antibiotics,’ but new research from Harvard scientists reveals concerning information about antibiotics, confirming that the antibiotic age coming to an end. Penicillin has been called better than the ‘big gun’ antibiotics for treating pneumonia and other childhood diseases, but is that really true in a new age of antibiotic resistance created by their overuse? Even the corrupt FDA admits that antibiotic misuse and overuse is a problem. According to the Harvard summary: “One of the oldest and most widely used antibiotics, penicillin, attacks enzymes that build the bacterial cell wall. Researchers have now shown that penicillin and its variants also set in motion a toxic malfunctioning of the cell’s wall-building machinery, dooming the cell to a futile cycle of building and then immediately destroying that wall.” This would be a simple microbial process that we could take for granted if it weren’t for the resistance to penicillin and other antibiotics that has emerged in the last few decades. The fact is that scientists still don’t really know how the original ‘age of antibiotics’ worked. Thomas Bernhardt, associate professor of microbiology and immunobiology at Harvard Medical School, are looking closer at this phenomenon. Their findings, published in the journal Cell, explain how penicillin can be devastating to bacteria — which may lead to new ways to thwart drug resistance, but could also explain why ‘good’ bacteria is harmed by antibiotics. How do these drugs differentiate after all? Bernhardt and his team have shown that antibiotic drugs do more than simply block cell-wall assembly. Penicillin and its variants also “set in motion a toxic malfunctioning of the cell’s wall-building machinery, which dooms the cell to a futile cycle of building and then immediately destroying that wall. This downstream death spiral depletes cells of the resources they need to survive.” Bernhardt explains the ramifications concerning drug-resistance: “It seems to be a common theme with some of the best antibiotics that we have: They don’t just inhibit the enzyme they are targeting; they actually convert that target so that whatever activity it has left becomes toxic.”
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